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Alzheimer's Disease: Return of the Prion Hypothesis
Recent experiments in mice have added weight to the idea that Alzheimer’s is driven by an infection-like spread of protein aggregates in the brain. Although it may seem startling, this hypothesis has been a popular one among Alzheimer’s researchers for the past several years (see “Alzheimer's Protein Shows Prion-like Infectiousness”). In fact, it represents a return to one of the first modern theories about Alzheimer’s: that it is like a weakly-transmissible prion disease.